Daisuke Sakai

Publication Activity (10 Years)

Years Active: 2021-2023
Publications (10 Years): 5

Top Topics

Vascular Endothelial Growth Factor

Preterm Infants

Cerebrospinal Fluid

Top Venues

Proceedings of the National Academy of Sciences of the United States of America

Molecular brain

International journal of molecular sciences

Journal of inflammation research

This page only lists publications with an associated author ORCID identifier.

Publications

Yuki Murakami, Yukio Imamura, Yoshiyuki Kasahara, Chihiro Yoshida, Yuta Momono, Ke Fang, Daisuke Sakai, Yukuo Konishi, Toshimasa Nishiyama
Maternal Inflammation with Elevated Kynurenine Metabolites Is Related to the Risk of Abnormal Brain Development and Behavioral Changes in Autism Spectrum Disorder. Cells 12 (7) (2023)
Sho Takata, Hiromi Sakata-Haga, Hiroki Shimada, Tsuyoshi Tsukada, Daisuke Sakai, Hiroki Shoji, Mitsuhiro Tomosugi, Yuka Nakamura, Yasuhito Ishigaki, Hideaki Iizuka, Yasuhiko Hayashi, Toshihisa Hatta
LIF-IGF Axis Contributes to the Proliferation of Neural Progenitor Cells in Developing Rat Cerebrum. International journal of molecular sciences 23 (21) (2022)
Karla T Falcon, Kristin E N Watt, Soma Dash, Ruonan Zhao, Daisuke Sakai, Emma L Moore, Sharien Fitriasari, Melissa Childers, Mihaela E Sardiu, Selene Swanson, Dai Tsuchiya, Jay R Uhruh, George Bugarinovic, Lin Li, Rita Shiang, Annita Achilleos, Jill Dixon, Michael J Dixon, Paul A Trainor
Dynamic regulation and requirement for ribosomal RNA transcription during mammalian development. Proceedings of the National Academy of Sciences of the United States of America 119 (31) (2022)
Daisuke Sakai, Takeru Sugawara, Tomonori Kurokawa, Yuki Murakami, Mitsuhiro Tomosugi, Hiroko Masuta, Hiromi Sakata-Haga, Toshihisa Hatta, Hiroki Shoji
Hif1α-dependent hypoxia signaling contributes to the survival of deep-layer neurons and cortex formation in a mouse model. Molecular brain 15 (1) (2022)
Yo Shimizu, Tsuyoshi Tsukada, Hiromi Sakata-Haga, Daisuke Sakai, Hiroki Shoji, Yutaka Saikawa, Toshihisa Hatta
Exposure to Maternal Immune Activation Causes Congenital Unfolded Protein Response Defects and Increases the Susceptibility to Postnatal Inflammatory Stimulation in Offspring. Journal of inflammation research 14 (2021)