Fei Shao

Publication Activity (10 Years)

Years Active: 2022-2024
Publications (10 Years): 13

Top Topics

Tyrosine Kinase

Epidermal Growth Factor Receptor

Cell Proliferation

Top Venues

Nature cell biology

Nature metabolism

This page only lists publications with an associated author ORCID identifier.

Publications

Leifeng Chen, Lin Zhang, Haihua He, Fei Shao, Zhentao Yu, Yibo Gao, Jie He
Ubiquitin-specific protease 54 regulates GLUT1-mediated aerobic glycolysis to inhibit lung adenocarcinoma progression by modifying p53 degradation. Oncogene 43 (26) (2024)
Juhong Wang, Yannan Yang, Fei Shao, Ying Meng, Dong Guo, Jie He, Zhimin Lu
Acetate reprogrammes tumour metabolism and promotes PD-L1 expression and immune evasion by upregulating c-Myc. Nature metabolism 6 (5) (2024)
Leifeng Chen, Lin Zhang, Haihua He, Fei Shao, Zhentao Yu, Yibo Gao, Jie He
Ubiquitin-specific protease 54 regulates GLUT1-mediated aerobic glycolysis to inhibit lung adenocarcinoma progression by modifying p53 degradation. Oncogene (2024)
Juhong Wang, Yannan Yang, Fei Shao, Ying Meng, Dong Guo, Jie He, Zhimin Lu
Acetate reprogrammes tumour metabolism and promotes PD-L1 expression and immune evasion by upregulating c-Myc. Nature metabolism (2024)
Juhong Wang, Yannan Yang, Fei Shao, Ying Meng, Dong Guo, Jie He, Zhimin Lu
Acetate reprogrammes tumour metabolism and promotes PD-L1 expression and immune evasion by upregulating c-Myc. Nature metabolism 6 (5) (2024)
Leifeng Chen, Lin Zhang, Haihua He, Fei Shao, Zhentao Yu, Yibo Gao, Jie He
Ubiquitin-specific protease 54 regulates GLUT1-mediated aerobic glycolysis to inhibit lung adenocarcinoma progression by modifying p53 degradation. Oncogene 43 (26) (2024)
Juhong Wang, Yannan Yang, Fei Shao, Ying Meng, Dong Guo, Jie He, Zhimin Lu
Acetate reprogrammes tumour metabolism and promotes PD-L1 expression and immune evasion by upregulating c-Myc. Nature metabolism 6 (5) (2024)
Tong Liu, Zheng Wang, Leiguang Ye, Yuran Duan, Hongfei Jiang, Haiyan He, Liwei Xiao, Qingang Wu, Yan Xia, Mengke Yang, Ke Wu, Meisi Yan, Guimei Ji, Yuli Shen, Lei Wang, Lin Li, Peixiang Zheng, Bofei Dong, Fei Shao, Xu Qian, Rilei Yu, Zhiren Zhang, Zhimin Lu, Daqian Xu
Nucleus-exported CLOCK acetylates PRPS to promote de novo nucleotide synthesis and liver tumour growth. Nature cell biology 25 (2) (2023)
Tong Liu, Zheng Wang, Leiguang Ye, Yuran Duan, Hongfei Jiang, Haiyan He, Liwei Xiao, Qingang Wu, Yan Xia, Mengke Yang, Ke Wu, Meisi Yan, Guimei Ji, Yuli Shen, Lei Wang, Lin Li, Peixiang Zheng, Bofei Dong, Fei Shao, Xu Qian, Rilei Yu, Zhiren Zhang, Zhimin Lu, Daqian Xu
Nucleus-exported CLOCK acetylates PRPS to promote de novo nucleotide synthesis and liver tumour growth. Nature cell biology 25 (2) (2023)
Tong Liu, Zheng Wang, Leiguang Ye, Yuran Duan, Hongfei Jiang, Haiyan He, Liwei Xiao, Qingang Wu, Yan Xia, Mengke Yang, Ke Wu, Meisi Yan, Guimei Ji, Yuli Shen, Lei Wang, Lin Li, Peixiang Zheng, Bofei Dong, Fei Shao, Xu Qian, Rilei Yu, Zhiren Zhang, Zhimin Lu, Daqian Xu
Nucleus-exported CLOCK acetylates PRPS to promote de novo nucleotide synthesis and liver tumour growth. Nature cell biology 25 (2) (2023)
Tong Liu, Zheng Wang, Leiguang Ye, Yuran Duan, Hongfei Jiang, Haiyan He, Liwei Xiao, Qingang Wu, Yan Xia, Mengke Yang, Ke Wu, Meisi Yan, Guimei Ji, Yuli Shen, Lei Wang, Lin Li, Peixiang Zheng, Bofei Dong, Fei Shao, Xu Qian, Rilei Yu, Zhiren Zhang, Zhimin Lu, Daqian Xu
Nucleus-exported CLOCK acetylates PRPS to promote de novo nucleotide synthesis and liver tumour growth. Nature cell biology 25 (2) (2023)
Tong Liu, Zheng Wang, Leiguang Ye, Yuran Duan, Hongfei Jiang, Haiyan He, Liwei Xiao, Qingang Wu, Yan Xia, Mengke Yang, Ke Wu, Meisi Yan, Guimei Ji, Yuli Shen, Lei Wang, Lin Li, Peixiang Zheng, Bofei Dong, Fei Shao, Xu Qian, Rilei Yu, Zhiren Zhang, Zhimin Lu, Daqian Xu
Nucleus-exported CLOCK acetylates PRPS to promote de novo nucleotide synthesis and liver tumour growth. Nature cell biology (2023)
Fei Shao, Yibo Gao, Wei Wang, Haiyan He, Liwei Xiao, Xiao Geng, Yan Xia, Dong Guo, Jing Fang, Jie He, Zhimin Lu
Silencing EGFR-upregulated expression of CD55 and CD59 activates the complement system and sensitizes lung cancer to checkpoint blockade. Nature cancer 3 (10) (2022)