Hideki Hayashi

Publication Activity (10 Years)

Years Active: 2022-2024
Publications (10 Years): 6

Top Topics

Venous Thromboembolism

Molecularly Imprinted

Reactive Oxygen Species

Top Venues

Proceedings of the National Academy of Sciences of the United States of America

International journal of molecular sciences

This page only lists publications with an associated author ORCID identifier.

Publications

Taisuke Nakamura, Mai Izumida, Manya Bakatumana Hans, Shuichi J Suzuki, Kensuke Takahashi, Hideki Hayashi, Koya Ariyoshi, Yoshinao Kubo
Post-Transcriptional Induction of the Antiviral Host Factor GILT/IFI30 by Interferon Gamma. International journal of molecular sciences 25 (17) (2024)
Hideki Hayashi, Tak Wah Mak, Yoshimasa Tanaka, Yoshinao Kubo, Mai Izumida, Ryuji Urae, Toshifumi Matsuyama
Development of a highly sensitive platform for protein-protein interaction detection and regulation of T cell function. Proceedings of the National Academy of Sciences of the United States of America 121 (33) (2024)
Hideki Hayashi, Tak Wah Mak, Yoshimasa Tanaka, Yoshinao Kubo, Mai Izumida, Ryuji Urae, Toshifumi Matsuyama
Development of a highly sensitive platform for protein-protein interaction detection and regulation of T cell function. Proceedings of the National Academy of Sciences of the United States of America 121 (33) (2024)
Hideki Hayashi, Tak Wah Mak, Yoshimasa Tanaka, Yoshinao Kubo, Mai Izumida, Ryuji Urae, Toshifumi Matsuyama
Development of a highly sensitive platform for protein-protein interaction detection and regulation of T cell function. Proceedings of the National Academy of Sciences of the United States of America 121 (33) (2024)
Mai Izumida, Osamu Kotani, Hideki Hayashi, Chris Smith, Tsutomu Fukuda, Koushirou Suga, Masatomo Iwao, Fumito Ishibashi, Hironori Sato, Yoshinao Kubo
Unique Mode of Antiviral Action of a Marine Alkaloid against Ebola Virus and SARS-CoV-2. Viruses 14 (4) (2022)
Yoshinao Kubo, Kiyoshi Yasui, Mai Izumida, Hideki Hayashi, Toshifumi Matsuyama
IDO1, FAT10, IFI6, and GILT Are Involved in the Antiretroviral Activity of γ-Interferon and IDO1 Restricts Retrovirus Infection by Autophagy Enhancement. Cells 11 (14) (2022)