Kathleen J Sweadner

ORCID

Publication Activity (10 Years)

Years Active: 2015-2024
Publications (10 Years): 6

Top Venues

This page only lists publications with an associated author ORCID identifier.

Publications

Yi Bessie Liu, Elena Arystarkhova, Amanda N Sacino, Margit V Szabari, Cathleen M Lutz, Markus Terrey, Natalia S Morsci, Tatjana C Jakobs, Karin Lykke-Hartmann, Allison Brashear, Elenora Napoli, Kathleen J Sweadner
Phenotype Distinctions in Mice Deficient in the Neuron-Specific α3 Subunit of Na,K-ATPase: Atp1a 3 tm1Ling/+ and Atp1a3 +/D801Y . eNeuro 11 (8) (2024)
Yi Bessie Liu, Elena Arystarkhova, Amanda N Sacino, Margit V Szabari, Cathleen M Lutz, Markus Terrey, Natalia S Morsci, Tatjana C Jakobs, Karin Lykke-Hartmann, Allison Brashear, Elenora Napoli, Kathleen J Sweadner
Phenotype Distinctions in Mice Deficient in the Neuron-Specific α3 Subunit of Na,K-ATPase: Atp1a 3 tm1Ling/+ and Atp1a3 +/D801Y . eNeuro 11 (8) (2024)
Yi Bessie Liu, Elena Arystarkhova, Amanda N Sacino, Margit V Szabari, Cathleen M Lutz, Markus Terrey, Natalia S Morsci, Tatjana C Jakobs, Karin Lykke-Hartmann, Allison Brashear, Elenora Napoli, Kathleen J Sweadner
Phenotype Distinctions in Mice Deficient in the Neuron-Specific α3 Subunit of Na,K-ATPase: Atp1a 3 tm1Ling/+ and Atp1a3 +/D801Y . eNeuro 11 (8) (2024)
Kyoko Hoshino, Kathleen J Sweadner, Toshitaka Kawarai, Jonas Alex Saute, Joel Freitas, Joana Damásio, Karina Carvalho Donis, Kazue Kimura, Hideki Fukuda, Masaharu Hayashi, Tetsuya Higuchi, Yoshio Ikeda, Laurie J Ozelius, Ryuji Kaji
Rapid-Onset Dystonia-Parkinsonism Phenotype Consistency for a Novel Variant of ATP1A3 in Patients Across 3 Global Populations. Neurology. Genetics 7 (2) (2021)
Elena Arystarkhova, Richard Bouley, Yi Bessie Liu, Kathleen J Sweadner
Impaired AQP2 trafficking in Fxyd1 knockout mice: A role for FXYD1 in regulated vesicular transport. PloS one 12 (11) (2017)
Yi Bessie Liu, Ambika Tewari, Johnny Salameh, Elena Arystarkhova, Thomas G Hampton, Allison Brashear, Laurie J Ozelius, Kamran Khodakhah, Kathleen J Sweadner
A dystonia-like movement disorder with brain and spinal neuronal defects is caused by mutation of the mouse laminin β1 subunit, Lamb1. eLife 4 (2015)

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Publication Activity (10 Years)

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Kathleen J Sweadner