Maja T Tomicic

Publication Activity (10 Years)

Years Active: 2018-2024
Publications (10 Years): 13

Top Topics

Oxidative Stress

Histone Deacetylase

Dna Damage Response

Top Venues

Nucleic acids research

Archives of toxicology

Cellular and molecular life sciences : CMLS

This page only lists publications with an associated author ORCID identifier.

Publications

Ariane Schmidt, Sebastian Allmann, Christian Schwarzenbach, Petra Snyder, Jia-Xuan Chen, Georg Nagel, Anna Schöneis, Birgit Rasenberger, Petra Beli, Alexander Loewer, Thomas G Hofmann, Maja T Tomicic, Markus Christmann
The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence. Nucleic acids research 52 (12) (2024)
Ariane Schmidt, Sebastian Allmann, Christian Schwarzenbach, Petra Snyder, Jia-Xuan Chen, Georg Nagel, Anna Schöneis, Birgit Rasenberger, Petra Beli, Alexander Loewer, Thomas G Hofmann, Maja T Tomicic, Markus Christmann
The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence. Nucleic acids research 52 (12) (2024)
Ariane Schmidt, Sebastian Allmann, Christian Schwarzenbach, Petra Snyder, Jia-Xuan Chen, Georg Nagel, Anna Schöneis, Birgit Rasenberger, Petra Beli, Alexander Loewer, Thomas G Hofmann, Maja T Tomicic, Markus Christmann
The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence. Nucleic acids research 52 (12) (2024)
Ariane Schmidt, Sebastian Allmann, Christian Schwarzenbach, Petra Snyder, Jia-Xuan Chen, Georg Nagel, Anna Schöneis, Birgit Rasenberger, Petra Beli, Alexander Loewer, Thomas G Hofmann, Maja T Tomicic, Markus Christmann
The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence. Nucleic acids research (2024)
Ariane Schmidt, Sebastian Allmann, Christian Schwarzenbach, Petra Snyder, Jia-Xuan Chen, Georg Nagel, Anna Schöneis, Birgit Rasenberger, Petra Beli, Alexander Loewer, Thomas G Hofmann, Maja T Tomicic, Markus Christmann
The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence. Nucleic acids research 52 (12) (2024)
Juliana B Vilar, Markus Christmann, Maja T Tomicic
Alterations in Molecular Profiles Affecting Glioblastoma Resistance to Radiochemotherapy: Where Does the Good Go? Cancers 14 (10) (2022)
Thomas R Reich, Christian Schwarzenbach, Juliana Brandstetter Vilar, Sven Unger, Fabian Mühlhäusler, Teodora Nikolova, Alicia Poplawski, H Irem Baymaz, Petra Beli, Markus Christmann, Maja T Tomicic
Localization matters: nuclear-trapped Survivin sensitizes glioblastoma cells to temozolomide by elevating cellular senescence and impairing homologous recombination. Cellular and molecular life sciences : CMLS 78 (14) (2021)
Maja T Tomicic, Mona Dawood, Thomas Efferth
Epigenetic Alterations Upstream and Downstream of p53 Signaling in Colorectal Carcinoma. Cancers 13 (16) (2021)
Christian Schwarzenbach, Larissa Tatsch, Juliana Brandstetter Vilar, Birgit Rasenberger, Lea Beltzig, Bernd Kaina, Maja T Tomicic, Markus Christmann
Targeting c-IAP1, c-IAP2, and Bcl-2 Eliminates Senescent Glioblastoma Cells Following Temozolomide Treatment. Cancers 13 (14) (2021)
Maja T Tomicic, Franziska Krämer, Alexandra Nguyen, Christian Schwarzenbach, Markus Christmann
Oxaliplatin-Induced Senescence in Colorectal Cancer Cells Depends on p14ARF-Mediated Sustained p53 Activation. Cancers 13 (9) (2021)
Maja T Tomicic, Christian Steigerwald, Birgit Rasenberger, Anamaria Brozovic, Markus Christmann
Functional mismatch repair and inactive p53 drive sensitization of colorectal cancer cells to irinotecan via the IAP antagonist BV6. Archives of toxicology 93 (8) (2019)
Christian Steigerwald, Birgit Rasenberger, Markus Christmann, Maja T Tomicic
Sensitization of colorectal cancer cells to irinotecan by the Survivin inhibitor LLP3 depends on XAF1 proficiency in the context of mutated p53. Archives of toxicology 92 (8) (2018)
Maja T Tomicic
Replica to the Opinion Letter regarding the article "Sensitization of colorectal cancer cells to irinotecan by the Survivin inhibitor LLP3 depends on XAF1 proficiency in the context of mutated p53" (Arch Toxicol https://doi.org/10.1007/s00204-018-240-x). Archives of toxicology 92 (10) (2018)