Matthias Lochner

ResearcherID ORCID

Publication Activity (10 Years)

Years Active: 2017-2022
Publications (10 Years): 5

Top Topics

Dendritic Cells

Immune Response

Toll Like Receptor

Top Venues

European journal of immunology

The Journal of experimental medicine

Nature communications

This page only lists publications with an associated author ORCID identifier.

Publications

Shuting Li, Chia-Wen Lu, Elia C Diem, Wang Li, Melanie Guderian, Marc Lindenberg, Friederike Kruse, Manuela Buettner, Stefan Floess, Markus R Winny, Robert Geffers, Hans Hermann Richnow, Wolf-Rainer Abraham, Guntram A Grassl, Matthias Lochner
Acetyl-CoA-Carboxylase 1-mediated de novo fatty acid synthesis sustains Lgr5 + intestinal stem cell function. Nature communications 13 (1) (2022)
Michael Beckstette, Chia-Wen Lu, Susanne Herppich, Elia C Diem, Anna Ntalli, Aaron Ochel, Friederike Kruse, Beate Pietzsch, Katrin Neumann, Jochen Huehn, Stefan Floess, Matthias Lochner
Profiling of epigenetic marker regions in murine ILCs under homeostatic and inflammatory conditions. The Journal of experimental medicine 219 (10) (2022)
Panagiota Mamareli, Friederike Kruse, Chia-Wen Lu, Melanie Guderian, Stefan Floess, Katharina Rox, David S J Allan, James R Carlyle, Mark Brönstrup, Rolf Müller, Luciana Berod, Tim Sparwasser, Matthias Lochner
Targeting cellular fatty acid synthesis limits T helper and innate lymphoid cell function during intestinal inflammation and infection. Mucosal immunology 14 (1) (2020)
Panagiota Mamareli, Friederike Kruse, Christin Friedrich, Nathiana Smit, Till Strowig, Tim Sparwasser, Matthias Lochner
Epithelium-specific MyD88 signaling, but not DCs or macrophages, control acute intestinal infection with Clostridium difficile. European journal of immunology 49 (5) (2019)
Christin Friedrich, Panagiota Mamareli, Sophie Thiemann, Friederike Kruse, Zuobai Wang, Bernhard Holzmann, Till Strowig, Tim Sparwasser, Matthias Lochner
MyD88 signaling in dendritic cells and the intestinal epithelium controls immunity against intestinal infection with C. rodentium. PLoS pathogens 13 (5) (2017)