Naoshi Kunimura

Publication Activity (10 Years)

Years Active: 2024-2024
Publications (10 Years): 5

Top Topics

Inflammatory Response

Oxidative Stress

Top Venues

Cancer research communications

This page only lists publications with an associated author ORCID identifier.

Publications

Daiki Kanaoka, Mitsuo Yamada, Hironori Yokoyama, Satoko Nishino, Naoshi Kunimura, Hiroshi Satoyoshi, Shota Wakabayashi, Kazunori Urabe, Takafumi Ishii, Masato Nakanishi
FPFT-2216, a novel anti-lymphoma compound, induces simultaneous degradation of IKZF1/3 and CK1α to activate p53 and inhibit NF-κB signaling. Cancer research communications (2024)
Daiki Kanaoka, Mitsuo Yamada, Hironori Yokoyama, Satoko Nishino, Naoshi Kunimura, Hiroshi Satoyoshi, Shota Wakabayashi, Kazunori Urabe, Takafumi Ishii, Masato Nakanishi
FPFT-2216, a Novel Anti-lymphoma Compound, Induces Simultaneous Degradation of IKZF1/3 and CK1α to Activate p53 and Inhibit NFκB Signaling. Cancer research communications 4 (2) (2024)
Daiki Kanaoka, Mitsuo Yamada, Hironori Yokoyama, Satoko Nishino, Naoshi Kunimura, Hiroshi Satoyoshi, Shota Wakabayashi, Kazunori Urabe, Takafumi Ishii, Masato Nakanishi
FPFT-2216, a Novel Anti-lymphoma Compound, Induces Simultaneous Degradation of IKZF1/3 and CK1α to Activate p53 and Inhibit NFκB Signaling. Cancer research communications 4 (2) (2024)
Daiki Kanaoka, Mitsuo Yamada, Hironori Yokoyama, Satoko Nishino, Naoshi Kunimura, Hiroshi Satoyoshi, Shota Wakabayashi, Kazunori Urabe, Takafumi Ishii, Masato Nakanishi
FPFT-2216, a Novel Anti-lymphoma Compound, Induces Simultaneous Degradation of IKZF1/3 and CK1α to Activate p53 and Inhibit NFκB Signaling. Cancer research communications 4 (2) (2024)
Daiki Kanaoka, Mitsuo Yamada, Hironori Yokoyama, Satoko Nishino, Naoshi Kunimura, Hiroshi Satoyoshi, Shota Wakabayashi, Kazunori Urabe, Takafumi Ishii, Masato Nakanishi
FPFT-2216, a novel anti-lymphoma compound, induces simultaneous degradation of IKZF1/3 and CK1α to activate p53 and inhibit NF-κB signaling. Cancer research communications (2024)