Nigel J Emptage (Nigel Emptage)

Publication Activity (10 Years)

Years Active: 2017-2024
Publications (10 Years): 5

Top Topics

Fluorescent Probe

Prefrontal Cortex

Top Venues

Philosophical transactions of the Royal Society of London. Series B, Biological sciences

Science signaling

This page only lists publications with an associated author ORCID identifier.

Publications

Carla C Schmidt, Rudi Tong, Nigel J Emptage
GluN2A- and GluN2B-containing pre-synaptic N -methyl-d-aspartate receptors differentially regulate action potential-evoked Ca 2+ influx via modulation of SK channels. Philosophical transactions of the Royal Society of London. Series B, Biological sciences 379 (1906) (2024)
Carla C Schmidt, Rudi Tong, Nigel J Emptage
GluN2A- and GluN2B-containing pre-synaptic N -methyl-d-aspartate receptors differentially regulate action potential-evoked Ca 2+ influx via modulation of SK channels. Philosophical transactions of the Royal Society of London. Series B, Biological sciences 379 (1906) (2024)
Carla C Schmidt, Rudi Tong, Nigel J Emptage
GluN2A- and GluN2B-containing pre-synaptic N -methyl-d-aspartate receptors differentially regulate action potential-evoked Ca 2+ influx via modulation of SK channels. Philosophical transactions of the Royal Society of London. Series B, Biological sciences 379 (1906) (2024)
William J Foster, Henry B C Taylor, Zahid Padamsey, Alexander F Jeans, Antony Galione, Nigel J Emptage
Hippocampal mGluR1-dependent long-term potentiation requires NAADP-mediated acidic store Ca2+ signaling. Science signaling 11 (558) (2018)
Zahid Padamsey, Rudi Tong, Nigel J Emptage
Glutamate is required for depression but not potentiation of long-term presynaptic function. eLife 6 (2017)