Phuong M Tran

Publication Activity (10 Years)

Years Active: 2018-2022
Publications (10 Years): 3

Top Topics

Pulmonary Arterial Hypertension

Staphylococcus Aureus

Machine Learning

Top Venues

Science signaling

Proceedings of the National Academy of Sciences of the United States of America

Science advances

University of Iowa

University of Iowa (PHD)

This page only lists publications with an associated author ORCID identifier.

Affiliation History

University of Iowa

United States

Graduate Research Assistant :: Microbiology and Immunology

University of Iowa May 04 2015

United States

Research Assistant :: Microbiology

Coe College

United States

Adjunct Instructor :: Biology

Coe College

United States

Adjunct Instructor :: Biology

University of Minnesota Duluth

United States

Graduate Teaching Assistant :: Biology

Education History

University of Iowa

United States

PHD :: Microbiology and Immunology

University of Minnesota Duluth

United States

MSC :: Integrated Biosciences

Publications

Kyle J Kinney, Sharon S Tang, Xiao-Jun Wu, Phuong M Tran, Nikhila S Bharadwaj, Katherine N Gibson-Corley, Ana N Forsythe, Katarina Kulhankova, Jenny E Gumperz, Wilmara Salgado-Pabón
SEC is an antiangiogenic virulence factor that promotes Staphylococcus aureus endocarditis independent of superantigen activity. Science advances 8 (19) (2022)
Nitija Tiwari, Marisa López-Redondo, Laura Miguel-Romero, Katarina Kulhankova, Michael P Cahill, Phuong M Tran, Kyle J Kinney, Samuel H Kilgore, Hassan Al-Tameemi, Christine A Herfst, Stephen W Tuffs, John R Kirby, Jeffrey M Boyd, John K McCormick, Wilmara Salgado-Pabón, Alberto Marina, Patrick M Schlievert, Ernesto J Fuentes
The SrrAB two-component system regulates Staphylococcus aureus pathogenicity through redox sensitive cysteines. Proceedings of the National Academy of Sciences of the United States of America 117 (20) (2020)
Michael S Zhang, Phuong M Tran, Alexander J Wolff, Mikaela M Tremblay, Micaela G Fosdick, Jon C D Houtman
Glycerol monolaurate induces filopodia formation by disrupting the association between LAT and SLP-76 microclusters. Science signaling 11 (528) (2018)