Sandra Kaiser

Publication Activity (10 Years)

Years Active: 2020-2024
Publications (10 Years): 7

Top Topics

Subarachnoid Hemorrhage

Top Venues

International journal of molecular sciences

Life (Basel, Switzerland)

This page only lists publications with an associated author ORCID identifier.

Publications

Sandra Kaiser, Luise Henrich, Iva Kiessling, Benedikt Loy, Nils Schallner
Neuroprotection via Carbon Monoxide Depends on the Circadian Regulation of CD36-Mediated Microglial Erythrophagocytosis in Hemorrhagic Stroke. International journal of molecular sciences 25 (3) (2024)
Sandra Kaiser, Luise Henrich, Iva Kiessling, Benedikt Loy, Nils Schallner
Neuroprotection via Carbon Monoxide Depends on the Circadian Regulation of CD36-Mediated Microglial Erythrophagocytosis in Hemorrhagic Stroke. International journal of molecular sciences 25 (3) (2024)
Sandra Kaiser, Luise Henrich, Iva Kiessling, Benedikt Loy, Nils Schallner
Neuroprotection via Carbon Monoxide Depends on the Circadian Regulation of CD36-Mediated Microglial Erythrophagocytosis in Hemorrhagic Stroke. International journal of molecular sciences 25 (3) (2024)
Sandra Kaiser, Luise Henrich, Iva Kiessling, Benedikt Loy, Nils Schallner
Neuroprotection via Carbon Monoxide Depends on the Circadian Regulation of CD36-Mediated Microglial Erythrophagocytosis in Hemorrhagic Stroke. International journal of molecular sciences 25 (3) (2024)
Sandra Kaiser, Luise Henrich, Iva Kiessling, Benedikt Loy, Nils Schallner
Neuroprotection via Carbon Monoxide Depends on the Circadian Regulation of CD36-Mediated Microglial Erythrophagocytosis in Hemorrhagic Stroke. International journal of molecular sciences 25 (3) (2024)
Sibylle Frase, Sandra Kaiser, Matti Steimer, Lisa Selzner, Niels Alexander Foit, Wolf-Dirk Niesen, Nils Schallner
Patients with Subarachnoid Hemorrhage Exhibit Disturbed Expression Patterns of the Circadian Rhythm Gene Period-2. Life (Basel, Switzerland) 11 (2) (2021)
Sandra Kaiser, Lisa Selzner, Janick Weber, Nils Schallner
Carbon monoxide controls microglial erythrophagocytosis by regulating CD36 surface expression to reduce the severity of hemorrhagic injury. Glia 68 (11) (2020)