Tamer M Gamal El-Din

Scopus Author ID ORCID

Publication Activity (10 Years)

Years Active: 2018-2024
Publications (10 Years): 21

Top Topics

Traumatic Brain Injury

Molecular Dynamics Simulations

Attention Deficit Hyperactivity Disorder

Structural Basis

Top Venues

Proceedings of the National Academy of Sciences of the United States of America

Nature cardiovascular research

Nature communications

This page only lists publications with an associated author ORCID identifier.

Publications

Ahmed Eltokhi, Brian Nils Lundstrom, Jin Li, Larry S Zweifel, William A Catterall, Tamer M Gamal El-Din
Pathogenic gating pore current conducted by autism-related mutations in the Na V 1.2 brain sodium channel. Proceedings of the National Academy of Sciences of the United States of America 121 (15) (2024)
Ahmed Eltokhi, Brian Nils Lundstrom, Jin Li, Larry S Zweifel, William A Catterall, Tamer M Gamal El-Din
Pathogenic gating pore current conducted by autism-related mutations in the Na V 1.2 brain sodium channel. Proceedings of the National Academy of Sciences of the United States of America 121 (15) (2024)
Lige Tonggu, Goragot Wisedchaisri, Tamer M Gamal El-Din, Michael J Lenaeus, Matthew M Logan, Tatsuya Toma, Justin Du Bois, Ning Zheng, William A Catterall
Dual receptor-sites reveal the structural basis for hyperactivation of sodium channels by poison-dart toxin batrachotoxin. Nature communications 15 (1) (2024)
Ahmed Eltokhi, Brian Nils Lundstrom, Jin Li, Larry S Zweifel, William A Catterall, Tamer M Gamal El-Din
Pathogenic gating pore current conducted by autism-related mutations in the Na V 1.2 brain sodium channel. Proceedings of the National Academy of Sciences of the United States of America 121 (15) (2024)
Lige Tonggu, Goragot Wisedchaisri, Tamer M Gamal El-Din, Michael J Lenaeus, Matthew M Logan, Tatsuya Toma, Justin Du Bois, Ning Zheng, William A Catterall
Dual receptor-sites reveal the structural basis for hyperactivation of sodium channels by poison-dart toxin batrachotoxin. Nature communications 15 (1) (2024)
Ahmed Eltokhi, Brian Nils Lundstrom, Jin Li, Larry S Zweifel, William A Catterall, Tamer M Gamal El-Din
Pathogenic gating pore current conducted by autism-related mutations in the Na V 1.2 brain sodium channel. Proceedings of the National Academy of Sciences of the United States of America 121 (15) (2024)
Lige Tonggu, Goragot Wisedchaisri, Tamer M Gamal El-Din, Michael J Lenaeus, Matthew M Logan, Tatsuya Toma, Justin Du Bois, Ning Zheng, William A Catterall
Dual receptor-sites reveal the structural basis for hyperactivation of sodium channels by poison-dart toxin batrachotoxin. Nature communications 15 (1) (2024)
Ahmed Eltokhi, Brian Nils Lundstrom, Jin Li, Larry S Zweifel, William A Catterall, Tamer M Gamal El-Din
Pathogenic gating pore current conducted by autism-related mutations in the Na V 1.2 brain sodium channel. Proceedings of the National Academy of Sciences of the United States of America 121 (15) (2024)
Ahmed Eltokhi, Brian Nils Lundstrom, Jin Li, Larry S Zweifel, William A Catterall, Tamer M Gamal El-Din
Pathogenic gating pore current conducted by autism-related mutations in the Na V 1.2 brain sodium channel. Proceedings of the National Academy of Sciences of the United States of America 121 (15) (2024)
Lige Tonggu, Goragot Wisedchaisri, Tamer M Gamal El-Din, Michael J Lenaeus, Matthew M Logan, Tatsuya Toma, Justin Du Bois, Ning Zheng, William A Catterall
Dual receptor-sites reveal the structural basis for hyperactivation of sodium channels by poison-dart toxin batrachotoxin. Nature communications 15 (1) (2024)
Ahmed Eltokhi, Brian Nils Lundstrom, Jin Li, Larry S Zweifel, William A Catterall, Tamer M Gamal El-Din
Pathogenic gating pore current conducted by autism-related mutations in the Na V 1.2 brain sodium channel. Proceedings of the National Academy of Sciences of the United States of America 121 (15) (2024)
Michael J Lenaeus, Tamer M Gamal El-Din, Lige Tonggu, Ning Zheng, William A Catterall
Structural basis for inhibition of the cardiac sodium channel by the atypical antiarrhythmic drug ranolazine. Nature cardiovascular research 2 (6) (2023)
Michael J Lenaeus, Tamer M Gamal El-Din, Lige Tonggu, Ning Zheng, William A Catterall
Structural basis for inhibition of the cardiac sodium channel by the atypical antiarrhythmic drug ranolazine. Nature cardiovascular research 2 (6) (2023)
Goragot Wisedchaisri, Tamer M Gamal El-Din, Natasha M Powell, Ning Zheng, William A Catterall
Structural basis for severe pain caused by mutations in the voltage sensors of sodium channel NaV1.7. The Journal of general physiology 155 (12) (2023)
Michael J Lenaeus, Tamer M Gamal El-Din, Lige Tonggu, Ning Zheng, William A Catterall
Structural basis for inhibition of the cardiac sodium channel by the atypical antiarrhythmic drug ranolazine. Nature cardiovascular research 2 (6) (2023)
Michael J Lenaeus, Tamer M Gamal El-Din, Lige Tonggu, Ning Zheng, William A Catterall
Structural basis for inhibition of the cardiac sodium channel by the atypical antiarrhythmic drug ranolazine. Nature cardiovascular research 2 (6) (2023)
Tamer M Gamal El-Din
When the Gates Swing Open Only: Arrhythmia Mutations That Target the Fast Inactivation Gate of Na v 1.5. Cells 11 (23) (2022)
Tamer M Gamal El-Din
When the Gates Swing Open Only: Arrhythmia Mutations That Target the Fast Inactivation Gate of Na v 1.5. Cells 11 (23) (2022)
Tamer M Gamal El-Din
When the Gates Swing Open Only: Arrhythmia Mutations That Target the Fast Inactivation Gate of Na v 1.5. Cells 11 (23) (2022)
Tamer M Gamal El-Din
When the Gates Swing Open Only: Arrhythmia Mutations That Target the Fast Inactivation Gate of Na v 1.5. Cells 11 (23) (2022)
Tamer M Gamal El-Din, Michael J Lenaeus, Karthik Ramanadane, Ning Zheng, William A Catterall
Molecular dissection of multiphase inactivation of the bacterial sodium channel NaVAb. The Journal of general physiology 151 (2) (2018)