Vanessa Aguiar-Pulido

Publication Activity (10 Years)

Years Active: 2021-2024
Publications (10 Years): 5

Top Topics

Induced Pluripotent Stem Cells

Dna Methylation

Top Venues

Proceedings of the National Academy of Sciences of the United States of America

Communications biology

This page only lists publications with an associated author ORCID identifier.

Publications

Stefan Wuchty, Alisa K White, Anouk M Olthof, Kyle Drake, Adam J Hume, Judith Olejnik, Vanessa Aguiar-Pulido, Elke Mühlberger, Rahul N Kanadia
Minor intron-containing genes as an ancient backbone for viral infection? PNAS nexus 3 (1) (2024)
Stefan Wuchty, Alisa K White, Anouk M Olthof, Kyle Drake, Adam J Hume, Judith Olejnik, Vanessa Aguiar-Pulido, Elke Mühlberger, Rahul N Kanadia
Minor intron-containing genes as an ancient backbone for viral infection? PNAS nexus 3 (1) (2024)
Xiao Han, Xuanye Cao, Vanessa Aguiar-Pulido, Wei Yang, Menuka Karki, Paula Andrea Pimienta Ramirez, Robert M Cabrera, Ying Linda Lin, Bogdan J Wlodarczyk, Gary M Shaw, Margaret Elizabeth Ross, Cui-Lian Zhang, Richard H Finnell, Yunping Lei
CIC missense variants contribute to susceptibility for spina bifida. Human mutation (2022)
Vanessa Aguiar-Pulido, Paul Wolujewicz, Alexander Martinez-Fundichely, Eran Elhaik, Gaurav Thareja, Alice Abdel Aleem, Nader Chalhoub, Tawny Cuykendall, Jamel Al-Zamer, Yunping Lei, Haitham El-Bashir, James M Musser, Abdulla Al-Kaabi, Gary M Shaw, Ekta Khurana, Karsten Suhre, Christopher E Mason, Olivier Elemento, Richard H Finnell, Margaret Elizabeth Ross
Systems biology analysis of human genomes points to key pathways conferring spina bifida risk. Proceedings of the National Academy of Sciences of the United States of America 118 (51) (2022)
Mariana Galvão Ferrarini, Avantika Lal, Rita Rebollo, Andreas J Gruber, Andrea Guarracino, Itziar Martinez Gonzalez, Taylor Floyd, Daniel Siqueira de Oliveira, Justin Shanklin, Ethan Beausoleil, Taneli Pusa, Brett E Pickett, Vanessa Aguiar-Pulido
Genome-wide bioinformatic analyses predict key host and viral factors in SARS-CoV-2 pathogenesis. Communications biology 4 (1) (2021)