Xue-Na Zhang

Publication Activity (10 Years)

Years Active: 2022-2024
Publications (10 Years): 6

Top Topics

Radiofrequency Ablation

High Fat Diet Induced

Top Venues

Hepatology communications

Proceedings of the National Academy of Sciences of the United States of America

Communications biology

This page only lists publications with an associated author ORCID identifier.

Publications

Ye Wang, Zhi-Hui Jiang, Yu-Wei Zhou, Tian-Tian Qiu, Han Wang, Min-Sheng Zhu, Xin Chen, Xue-Na Zhang
Gallbladder dysfunction caused by MYPT1 ablation triggers cholestasis-induced hepatic fibrosis in mice. Hepatology communications 8 (7) (2024)
Ye Wang, Zhi-Hui Jiang, Yu-Wei Zhou, Tian-Tian Qiu, Han Wang, Min-Sheng Zhu, Xin Chen, Xue-Na Zhang
Gallbladder dysfunction caused by MYPT1 ablation triggers cholestasis-induced hepatic fibrosis in mice. Hepatology communications 8 (7) (2024)
Ye Wang, Zhi-Hui Jiang, Yu-Wei Zhou, Tian-Tian Qiu, Han Wang, Min-Sheng Zhu, Xin Chen, Xue-Na Zhang
Gallbladder dysfunction caused by MYPT1 ablation triggers cholestasis-induced hepatic fibrosis in mice. Hepatology communications 8 (7) (2024)
Ye Wang, Zhi-Hui Jiang, Yu-Wei Zhou, Tian-Tian Qiu, Han Wang, Min-Sheng Zhu, Xin Chen, Xue-Na Zhang
Gallbladder dysfunction caused by MYPT1 ablation triggers cholestasis-induced hepatic fibrosis in mice. Hepatology communications 8 (7) (2024)
Yu-Wei Zhou, Jie Sun, Ye Wang, Cai-Ping Chen, Tao Tao, Ming Ma, Xin Chen, Xue-Na Zhang, Li-Yuan Yang, Zhong-Liang Zhang, Ye-Qiong Li, Zhi-Hui Jiang, Tian-Tian Qiu, Han Wang, Yang Pan, Jian Zhang, Hua-Qun Chen, Pei Wang, Min-Sheng Zhu
Tas2R activation relaxes airway smooth muscle by release of Gα t targeting on AChR signaling. Proceedings of the National Academy of Sciences of the United States of America 119 (26) (2022)
Wei Zhao, Jie Sun, Liang-Yu Yao, Dong Hang, Ye-Qiong Li, Cai-Ping Chen, Yu-Wei Zhou, Xin Chen, Tao Tao, Li-Sha Wei, Yan-Yan Zheng, Xie Ge, Chao-Jun Li, Zhong-Cheng Xin, Yang Pan, Xin-Zhu Wang, Wei-Qi He, Xue-Na Zhang, Bing Yao, Min-Sheng Zhu
MYPT1 reduction is a pathogenic factor of erectile dysfunction. Communications biology 5 (1) (2022)