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Compensatory post-diuretic renal sodium reabsorption is not a dominant mechanism of diuretic resistance in acute heart failure.

Zachary L CoxVeena S RaoJuan Betuel Ivey-MirandaJulieta Moreno-VillagómezDevin MahoneyPiotr PonikowskiJan BiegusJeffrey M TurnerChristopher MaulionLavanya BellumkondaJennifer L AsherHelen PariseFrancis Perry WilsonDavid H EllisonChristopher S WilcoxJeffrey M Testani
Published in: European heart journal (2021)
On a population level, CPDSR was not an important driver of diuretic resistance in hypervolemic ADHF. Contrary to CPDSR, a greater diuretic-induced natriuresis predicted a larger post-diuretic spontaneous natriuresis. Basal sodium avidity, rather than diuretic-induced CPDSR, appears to be the predominant determinate of both diuretic-induced and post-diuretic natriuresis in hypervolemic ADHF.
Keyphrases
  • acute heart failure
  • heart failure
  • high glucose
  • diabetic rats
  • endothelial cells