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ONC201 in Combination with Paxalisib for the Treatment of H3K27-Altered Diffuse Midline Glioma.

Evangeline R JacksonRyan J DuchatelDilana E StaudtMika L PerssonAbdul MannanSridevi YadavilliSarah ParackalShaye GameWai Chin ChongW Samantha Nilanthi JayasekaraMarion Le GrandPadraic S KearneyAlicia M DouglasIzac J FindlayZacary P GermonHolly P McEwenTyrone S BeitakiAdjanie PatabendigeDavid A Skerrett-ByrneBrett NixonNathan D SmithBryan W DayNeevika ManoharanSumanth NagabushanJordan R HansfordDinisha GovenderGeoffrey B McCowageRon FiresteinMeegan HowlettRaelene EndersbyNicholas G GottardoFrank AlvaroSebastian M WaszakMartin Røssel LarsenYolanda Colino-SanguinoFatima Valdes MoraAndria RakotomalalaSamuel MeignanEddy PasquierAnne Isabelle BertozziEsther HullemanDavid D EisenstatNicholas A VitanzaJavad NazarianCarl J KoschmannRomain MuellerJason E CainMatthew D Dun
Published in: Cancer research (2023)
PI3K/Akt signaling promotes metabolic adaptation to ONC201-mediated disruption of mitochondrial energy homeostasis in diffuse intrinsic pontine glioma, highlighting the utility of a combination treatment strategy using ONC201 and the PI3K/Akt inhibitor paxalisib.
Keyphrases
  • pi k akt
  • signaling pathway
  • cell proliferation
  • low grade
  • oxidative stress
  • cell death
  • cell cycle arrest