Galectin-3 in Atrial Fibrillation: Mechanisms and Therapeutic Implications.
Nicolas ClementyEric PiverArnaud BissonClémentine AndreAnne BernardBertrand PierreLaurent FauchierDominique BabutyPublished in: International journal of molecular sciences (2018)
Maintenance of atrial fibrillation is a complex mechanism, including extensive electrical and structural remodeling of the atria which involves progressive fibrogenesis. Galectin-3 is a biomarker of fibrosis, and, thus, may be involved in atrial remodeling in atrial fibrillation patients. We review the role of galectin-3 in AF mechanisms and its potential therapeutic implications.
Keyphrases
- atrial fibrillation
- catheter ablation
- left atrial
- oral anticoagulants
- left atrial appendage
- direct oral anticoagulants
- end stage renal disease
- heart failure
- ejection fraction
- percutaneous coronary intervention
- newly diagnosed
- multiple sclerosis
- chronic kidney disease
- peritoneal dialysis
- venous thromboembolism
- coronary artery disease
- acute coronary syndrome
- left ventricular
- patient reported