Activation of Laminin γ2 by Helicobacter pylori promotes invasion and survival of gastric cancer cells with E-cadherin defects.

Rui Manuel FerreiraJoana FigueiredoInes Pinto-RibeiroIrene GulloDionyssios N SgourasLaura CarretoPatricia CastroManuel A SantosFatima CarneiroRaquel SerucaCeu Figueiredo

Published in: The Journal of infectious diseases (2022)

These data highlight Laminin γ2 and its downstream effectors as potential therapeutic targets, and the value of H. pylori eradication to delay gastric cancer onset and progression.

Keyphrases

helicobacter pylori
helicobacter pylori infection
electronic health record
cell migration
big data
free survival
type iii
machine learning
data analysis
risk assessment
artificial intelligence