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Transcriptomic analysis reveals the role of SIX1 in mouse cranial neural crest patterning and bone development.

Aparna BaxiKaryn JourdeuilTimothy C CoxDavid E ClouthierAndre L P Tavares
Published in: Developmental dynamics : an official publication of the American Association of Anatomists (2023)
Our results clarify the mechanisms by which SIX1 regulates the development of NC-derived craniofacial elements that are altered in SIX1-associated disorders. In addition, this work identifies novel genes that could be causative to this birth defect and establishes a link between SIX1 and WNT signaling during patterning of NC cells.
Keyphrases
  • genome wide
  • induced apoptosis
  • cell cycle arrest
  • bone mineral density
  • dna methylation
  • cell fate
  • cell death
  • pregnant women
  • gestational age
  • transcription factor
  • genome wide identification
  • bone regeneration