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Lung Cancer in Ever- and Never-Smokers: Findings from Multi-Population GWAS Studies.

Yafang LiXiangjun XiaoJian-Rong LiYounghun HanChao ChengGail F FernandesShannon E SlewitzkeSusan M RosenbergMeng ZhuJinyoung ByunYohan BosseJames D McKayDemetrios AlbanesStephen C LamAdonina TardónChu ChenStig Egil BojesenMaria Teresa LandiMattias J JohanssonAngela RischHeike BickeböllerHeinz-Erich WichmannDavid Chistopher ChristianiGadi RennertSusanne M ArnoldGary E GoodmanJohn K FieldMichael P A DaviesSanjay S SheteLoic Le MarchandGeoffrey LiuRayjean J HungAngeline S AndrewLambertus A L M KiemeneyRyan SunShanbeh Zienolddiny-NaruiKjell GrankvistMikael JohanssonNeil C CaporasoAngela CoxYun Chul HongPhilip LazarusMatthew B SchabathMelinda C AldrichKendra L SchwartzIvan P GorlovKristen S PurringtonPing YangYanhong LiuRichard K WilsonSusan M PinneyDiptasri MandalJames C WilleyColette GabaPaul J BrennanJun XiaHongbing ShenChristopher Ian Amos
Published in: Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology (2024)
Our study highlights the genetic heterogeneity between ever- and never-smoking lung cancer and provides etiologic insights into the complicated genetic architecture of this deadly cancer.
Keyphrases
  • smoking cessation
  • genome wide
  • papillary thyroid
  • copy number
  • single cell
  • squamous cell
  • gene expression
  • lymph node metastasis