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CBP/P300 BRD Inhibition Reduces Neutrophil Accumulation and Activates Antitumor Immunity in TNBC.

Xueying YuanXiaoxin HaoHilda L ChanNa ZhaoDiego A PedrozaFengshuo LiuKang LeAlex J SmithSebastian J CalderonNadia LieuMichael J SothPhilip JonesXiang H F ZhangJeffrey M Rosen
Published in: bioRxiv : the preprint server for biology (2024)
In neutrophil-enriched triple-negative breast cancer (TNBC) models, CREB binding protein (CBP)/P300 bromodomain (BRD) inhibition reduces tumor growth and systemic neutrophil accumulation while stimulating an antitumor immune response. This improves standard-of-care therapies, suggesting a potential therapeutic benefit of CBP/P300 BRD inhibitors for neutrophil-enriched TNBC.
Keyphrases
  • immune response
  • binding protein
  • healthcare
  • palliative care
  • pain management