Genetic variations in ATM and H2AX loci contribute to risk of hematological abnormalities in individuals exposed to BTEX chemicals.

Samaneh Jafari RoshanYaser MansooriSeyed Reza HosseiniDavood SabourAbdolreza Daraei

Published in: Journal of clinical laboratory analysis (2022)

These observations may help to understand the mechanisms of BTEX hematotoxicity and identify useful biomarkers of risk assessment for workers exposed to BTEX.

Keyphrases

risk assessment
genome wide
dna damage
dna repair
dna methylation
copy number
gene expression
oxidative stress