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Tollip interaction with STAT3: a novel mechanism to regulate human airway epithelial responses to type 2 cytokines.

Niccolette SchaunamanKris Genelyn DimasuayMonica KraftHong Wei Chu
Published in: Respiratory research (2022)
Our results, for the first time, suggest that Tollip inhibits excessive eotaxin-3 induction by IL-13, in part through the interaction and inhibition of STAT3. These findings lend evidence to the potential of a STAT3 inhibitor as a therapeutic target, especially for type 2 inflammation-high asthmatics with Tollip deficiency.
Keyphrases
  • cell proliferation
  • endothelial cells
  • oxidative stress
  • human health