Bioactive Sphingolipids, Complement Cascade, and Free Hemoglobin Levels in Stable Coronary Artery Disease and Acute Myocardial Infarction.
Tomasz JadczykK BaranskiM SyzdolE NabialekW WanhaRadoslaw KurzelowskiM Z RatajczakM KuciaBarbara DolegowskaM NiewczasJ ZejdaWojciech WojakowskiPublished in: Mediators of inflammation (2018)
Inflammatory response after AMI and CABG differed regarding the release of sphingolipids, free hemoglobin, and complement cascade cleavage fragments.
Keyphrases
- acute myocardial infarction
- percutaneous coronary intervention
- inflammatory response
- coronary artery disease
- coronary artery bypass grafting
- left ventricular
- lipopolysaccharide induced
- red blood cell
- lps induced
- coronary artery bypass
- acute coronary syndrome
- cardiovascular events
- toll like receptor
- cardiovascular disease
- heart failure
- type diabetes
- dna binding
- transcatheter aortic valve replacement
- tissue engineering
- aortic stenosis