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Aryl Hydrocarbon Receptor Knockout Accelerates PanIN Formation and Fibro-Inflammation in a Mutant Kras -Driven Pancreatic Cancer Model.

Morgan T WalcheckPatrick B SchwartzNoah D CarrilloKristina A MatkowskyjManabu NukayaChristopher A BradfieldSean M Ronnekleiv-Kelly
Published in: Pancreas (2024)
These findings show the loss of AHR results in heightened Kras -induced PanIN formation, through modulation of immune cells within the pancreatic tumor microenvironment.
Keyphrases
  • wild type
  • oxidative stress
  • high glucose
  • diabetic rats
  • drug induced
  • endothelial cells
  • binding protein