Acute hyperventilation increases the central venous-to-arterial PCO2 difference in stable septic shock patients.

Jihad MallatUsman MohammadMalcolm LemyzeMehdi MeddourMarie JonardFlorent PepyGaelle GasanStephanie BarraillerJohanna TemimeNicolas VangrunderbeeckLaurent TronchonDidier Thevenin

Published in: Annals of intensive care (2017)

Acute hyperventilation provoked a significant increase in ∆PCO2, which was the result of a significant increase in VO2 induced by hyperventilation. The clinician should be aware of the effects of acute elevation of alveolar ventilation on ∆PCO2.

Keyphrases

respiratory failure
liver failure
septic shock
drug induced
aortic dissection
end stage renal disease
newly diagnosed
extracorporeal membrane oxygenation
hepatitis b virus
prognostic factors
intensive care unit
patient reported outcomes
acute respiratory distress syndrome