Nicotine Potentially Alters Endothelial Inflammation and Cell Adhesion via LGALS9 .

Sönke Maximilian BraßAgnesa Mazrekaj Joscha MulorzWiebke IbingKim-Jürgen KrottKiku TakeuchiMelanie CappalloHsiang-Han LiuMargitta ElversHubert SchelzigMarkus Udo Wagenhäuser

Published in: Journal of cardiovascular development and disease (2023)

itself may serve as key regulator for essential endothelial cell processes via interfering with various signaling pathways and may thus represent a potentially novel target in the pathogenesis of aortic pathologies.

Keyphrases

cell adhesion
endothelial cells
signaling pathway
oxidative stress
aortic valve
smoking cessation
transcription factor
high glucose
pulmonary artery
pi k akt
heart failure
epithelial mesenchymal transition
atrial fibrillation