ER stress promotes HBV production by enhancing utilization of the autophagosome- multivesicular body axis.

Xueyu WangZhiqiang WeiBin ChengJia LiYulin HeTingyu LanThekla KemperYong LinBin JiangYongfang JiangZhongji MengMengji Lu

Published in: Hepatology (Baltimore, Md.) (2021)

TM-induced ER stress and autophagic flux promoted HBV replication and the release of SVPs and naked capsids through the autophagosome-late endosome/MVB axis.

Keyphrases

hepatitis b virus
liver failure
cell death
high glucose
drug induced