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β cell acetate production and release are negligible.

Kai XuChioma NnyamahNupur PandyaNadia SweisIrene Corona-AvilaMedha PriyadarshiniBarton WicksteedBrian T Layden
Published in: Islets (2024)
Our findings do not substantiate the glucose-dependent release of acetate from pancreatic β cells, and therefore, invalidate the possibility of an autocrine inhibitory effect on glucose stimulated insulin secretion.
Keyphrases
  • induced apoptosis
  • blood glucose
  • cell cycle arrest
  • single cell
  • cell therapy
  • stem cells
  • oxidative stress
  • type diabetes
  • adipose tissue
  • skeletal muscle
  • pi k akt