Mitochondrial-targeted antioxidant ingestion acutely blunts VO 2max in physically inactive females.
Ryan P HughesNicholas A CarliniBradley S FleenorMatthew P HarberPublished in: Physiological reports (2023)
, which was primarily mediated by impairment of ventilatory function. These data suggest that the acute accumulation of exercise-induced mitochondrial reactive oxygen species (mtROS) are necessary for maximal aerobic capacity. Further research is warranted on mtROS-antioxidant cell signaling cascades, and how they relate to mitochondrial function during exercise.
Keyphrases
- oxidative stress
- reactive oxygen species
- high intensity
- resistance training
- liver failure
- anti inflammatory
- single cell
- electronic health record
- cell therapy
- physical activity
- respiratory failure
- heart rate
- cancer therapy
- big data
- drug induced
- aortic dissection
- blood pressure
- stem cells
- body composition
- bone marrow
- artificial intelligence
- deep learning
- acute respiratory distress syndrome
- mechanical ventilation