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Mitochondrial ROS control neuronal excitability and cell fate in frontotemporal dementia.

Noemí EsterasOlga KopachMarta MaiolinoVincenzo LaricciaSalvatore AmorosoSeema QamarSelina WrayDmitri A RusakovMorana JaganjacAndrey Y Abramov
Published in: Alzheimer's & dementia : the journal of the Alzheimer's Association (2021)
These results demonstrate mitochondrial ROS modulate glutamatergic signaling in FTD, and suggest a new therapeutic strategy.
Keyphrases
  • cell fate
  • oxidative stress
  • dna damage
  • cell death
  • reactive oxygen species
  • brain injury
  • subarachnoid hemorrhage