The role of anti-citrullinated protein antibody in pathogenesis of RA.
Hang MaXu LiangShan-Shan LiWei LiTian-Fang LiPublished in: Clinical and experimental medicine (2024)
Rheumatoid arthritis (RA) is a common autoimmune rheumatic disease that causes chronic synovitis, bone erosion, and joint destruction. The autoantigens in RA include a wide array of posttranslational modified proteins, such as citrullinated proteins catalyzed by peptidyl arginine deiminase4a. Pathogenic anti-citrullinated protein antibodies (ACPAs) directed against a variety of citrullinated epitopes are abundant both in plasma and synovial fluid of RA patients. ACPAs play an important role in the onset and progression of RA. Intensive and extensive studies are being conducted to unveil the mechanisms of RA pathogenesis and evaluate the efficacy of some investigative drugs. In this review, we focus on the formation and pathogenic function of ACPAs.
Keyphrases
- rheumatoid arthritis
- disease activity
- ankylosing spondylitis
- interstitial lung disease
- end stage renal disease
- systemic lupus erythematosus
- ejection fraction
- chronic kidney disease
- nitric oxide
- peritoneal dialysis
- amino acid
- high resolution
- bone mineral density
- drug induced
- prognostic factors
- high throughput
- postmenopausal women
- idiopathic pulmonary fibrosis
- case control