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Mutant Phosphodiesterase 3A Protects From Hypertension-Induced Cardiac Damage.

Maria ErcuMichael B MückeTamara PallienLajos MarkóAnastasiia SholokhCarolin SchächterleAtakan AydinAlexa KiddStephan WalterYasmin EsmatiBrandon J McMurrayDaniella F LatoDaniele Yumi Sunaga-FranzePhilip H DierksBarbara Isabel Montesinos FloresRyan Walker-GrayMaolian GongClaudia MerticariuKerstin ZühlkeMichael RusswurmTiannan LiuTheda Ulrike Patricia BartolomaeusSabine PautzStefanie SchelenzMartin TaubeHanna NapieczynskaArnd HeuserJenny EichhorstMartin LehmannDuncan C MillerSebastian DieckeFatimunnisa QadriElena PopovaReika LangankiMatthew A MovsesianFriedrich W HerbergSofia K ForslundDominik N MullerTatiana BorodinaPhilipp G MaassSylvia BähringNorbert HübnerMichael BaderAnastasiia Sholokh
Published in: Circulation (2022)
mutations in the heart rather than nonselective PDE3 inhibition is cardioprotective in the long term. Our findings could facilitate the search for new treatments to prevent hypertension-induced cardiac damage.
Keyphrases
  • stress induced
  • blood pressure
  • high glucose
  • diabetic rats
  • oxidative stress
  • heart failure
  • drug induced
  • arterial hypertension