USP38 exacerbates atrial inflammation, fibrosis, and susceptibility to atrial fibrillation after myocardial infarction in mice.

Yang GongTingting YuWei ShuaiTao ChenJingjing ZhangHe Huang

Published in: Molecular medicine (Cambridge, Mass.) (2023)

USP38 plays an important role in atrial inflammation, fibrosis, and AF susceptibility after MI, providing a promising target for the treatment of AF after MI.

Keyphrases

atrial fibrillation
catheter ablation
left atrial
oxidative stress
oral anticoagulants
left atrial appendage
direct oral anticoagulants
heart failure
percutaneous coronary intervention
high fat diet induced
acute coronary syndrome
venous thromboembolism
left ventricular
wild type