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Endothelial YAP/TEAD1-CXCL17 signaling recruits myeloid-derived suppressor cells against liver ischemia-reperfusion injury.

Sitong ZhangZhongquan SunZhenhua ChenYanli BiShenyu WeiZhengwei MaoJin JinYuan DingWei-Lin Wang
Published in: Hepatology (Baltimore, Md.) (2024)
Endothelial YAP/TEAD1-CXCL17 signaling recruited MDSCs to attenuate liver IRI, providing evidence of a therapeutic potential for managing IRI in liver surgery.
Keyphrases
  • ischemia reperfusion injury
  • endothelial cells
  • induced apoptosis
  • oxidative stress
  • cell cycle arrest
  • cell death
  • signaling pathway