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Tunicamycin-Induced Endoplasmic Reticulum Stress Damages Complex I in Cardiac Mitochondria.

Qun ChenJeremy ThompsonYing HuEdward J Lesnefsky
Published in: Life (Basel, Switzerland) (2022)
Induction of the ER stress using TUNI contributes to complex I damage by activating calpain 1.
Keyphrases
  • endoplasmic reticulum stress
  • induced apoptosis
  • signaling pathway
  • oxidative stress
  • high glucose
  • diabetic rats
  • left ventricular
  • heart failure
  • drug induced
  • reactive oxygen species
  • stress induced