Atopic dermatitis: Interaction between genetic variants of GSTP1, TNF, TLR2, and TLR4 and air pollution in early life.
Anke HülsClaudia KlümperElaina A MacIntyreMichael BrauerErik MelénMario BauerDietrich BerdelAnna BergströmBert BrunekreefMoira Chan-YeungElaine FuertesUlrike GehringAnna GrefJoachim HeinrichMarie StandlIrina LehmannMarjan KerkhofGerard H KoppelmanAnita L KozyrskyjGöran PershagenChristopher CarlstenUrsula KrämerTamara Schikowskinull nullPublished in: Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology (2018)
The marginal genetic association of a weighted GRS from GSTP1, TNF, TLR2, and TLR4SNPs and its interaction with air pollution supports the role of oxidative stress and inflammation in AD.
Keyphrases
- toll like receptor
- air pollution
- inflammatory response
- oxidative stress
- early life
- immune response
- rheumatoid arthritis
- atopic dermatitis
- nuclear factor
- genome wide
- particulate matter
- dna damage
- magnetic resonance
- lung function
- magnetic resonance imaging
- ischemia reperfusion injury
- gene expression
- diabetic rats
- copy number