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R-spodin2 enhances canonical Wnt signaling to maintain the stemness of glioblastoma cells.

Si LiuKin Pong UJieting ZhangLai Ling TsangJiawei HuangShui Ping TuXiaohua Jiang
Published in: Cancer cell international (2018)
Our study reveals a novel regulatory mechanisms underlying the over-activation of β-catenin-mediated signaling in the pathogenesis of GBM.
Keyphrases
  • epithelial mesenchymal transition
  • induced apoptosis
  • stem cells
  • cell cycle arrest
  • cell proliferation
  • transcription factor
  • oxidative stress
  • cancer stem cells