Inhibition of IAP (inhibitor of apoptosis) proteins represses inflammatory status via nuclear factor-kappa B pathway in murine endometriosis lesions.

Fuminori TaniguchiTakashi UegakiKazuomi NakamuraKhine Yin MonTakashi HaradaTetsuya OhbayashiTasuku Harada

Published in: American journal of reproductive immunology (New York, N.Y. : 1989) (2017)

Inhibitor of apoptosis proteins antagonist represses the inflammation status of murine endometriosis-like lesions viaNF-κB pathway. IAPs may be a novel therapeutic target for endometriosis.

Keyphrases

nuclear factor
oxidative stress
toll like receptor
endoplasmic reticulum stress
cell cycle arrest
cell death
immune response