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Receptor-interacting protein kinase 2 contributes to host innate immune responses against Fusobacterium nucleatum in macrophages and decidual stromal cells.

Ji-Yeon ParkTae-Sung LeeEui Jeong NohAh-Ra JangJae-Hun AhnDong-Yeon KimDo-Hyeon JungEun-Jung SongYeon-Ji LeeYun-Ji LeeSung Ki LeeJong-Hwan Park
Published in: American journal of reproductive immunology (New York, N.Y. : 1989) (2021)
In conclusion, Ripk2 signaling appears to contribute to the F. nucleatum-induced immune response and can be a preventive and therapeutic target against APOs.
Keyphrases
  • innate immune
  • protein kinase
  • immune response
  • high glucose
  • diabetic rats
  • drug induced
  • toll like receptor
  • oxidative stress