Exogenous vasopressin dose-dependently modulates gastric microcirculatory oxygenation in dogs via V1A receptor.
Richard TruseSteven GreweAnna HerminghausJan SchulzAndreas P M WeberTabea Mettler-AltmannInge BauerOlaf PickerChristian VollmerPublished in: Critical care (London, England) (2019)
Exogenous AVP dose-dependently modulates gastric μHbO2, with an increased μHbO2 with ultra-low dose AVP. The effects of AVP on μHbO2 are abolished by V1A receptor inhibition. These effects are independent of a modulation of systemic hemodynamic variables.