COVID-19-Induced Thrombosis in Patients without Gastrointestinal Symptoms and Elevated Fecal Calprotectin: Hypothesis Regarding Mechanism of Intestinal Damage Associated with COVID-19.
Mauro GiuffréStefano di BellaGianluca SambataroVerena ZerbatoMarco CavallaroAlessandro Agostino OcchipintiAndrea PalermoAnna CrescentiFabio MonicaRoberto LuzzatiLory Saveria CrocèPublished in: Tropical medicine and infectious disease (2020)
our findings may change the current understanding of COVID-19 intestinal-related disease pathogenesis, shedding new light on the potential role of thrombosis and the consequent hypoxic intestinal damage.
Keyphrases
- coronavirus disease
- sars cov
- end stage renal disease
- oxidative stress
- ejection fraction
- newly diagnosed
- chronic kidney disease
- respiratory syndrome coronavirus
- prognostic factors
- diabetic rats
- high glucose
- patient reported outcomes
- drug induced
- rheumatoid arthritis
- risk assessment
- systemic lupus erythematosus
- disease activity
- endothelial cells
- human health