Association of Maternal Air Pollution Exposure and Infant Lung Function Is Modified by Genetic Propensity to Oxidative Stress.
Dwan VilcinsWen Ray LeeCindy PhamSam TannerLuke D KnibbsDavid P BurgnerTamara L BlakeToby MansellAnne-Louise PonsonbyPeter D Slynull Barwon Infant Study Investigator GroupPublished in: Children (Basel, Switzerland) (2024)
is associated with a lower FRC and a higher LCI score in infants with a genetic propensity to oxidative stress. There was no relationship between maternal exposure to air pollution with maternal and cord blood inflammation or OS biomarkers.
Keyphrases
- air pollution
- lung function
- oxidative stress
- cord blood
- birth weight
- particulate matter
- pregnancy outcomes
- cystic fibrosis
- chronic obstructive pulmonary disease
- dna damage
- ischemia reperfusion injury
- genome wide
- diabetic rats
- induced apoptosis
- copy number
- weight gain
- pregnant women
- body mass index
- gestational age
- endoplasmic reticulum stress