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The extracellular matrix molecules versican and hyaluronan in urethral and vaginal tissues in stress urinary incontinence.

Ingrid A HartenStephen P EvankoChong H ChoeEugene W LeeBhavin N PatelMarika BogdaniThomas N WightUna J Lee
Published in: Neurourology and urodynamics (2021)
Reduction of Vcan-degrading enzymes and HA synthases at day 4 postsurgery indicates a potential delay in ECM turnover associated with SUI. Abundant Vcan is associated with inflammation and elastin fiber network disruption, warranting further investigation to determine its role in SUI pathogenesis.
Keyphrases
  • extracellular matrix
  • oxidative stress
  • gene expression
  • bone mineral density
  • urinary incontinence
  • risk assessment