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Rare Variants in Complement Gene in C3 Glomerulopathy and Immunoglobulin-Mediated Membranoproliferative Glomerulonephritis.

Marie-Sophie MeulemanPaula Vieira MartinsCarine El SissyVincent AudardVeronique BaudouinDominique BertrandFrank BridouxFérielle LouilletClaire DossierVincent L M EsnaultNoémie Jourde-ChicheAlexandre KarrasMarie-Pascale MorinFrançois ProvotPhilippe RemyDavid RibesCaroline Rousset-RouviereAude ServaisEric ThervetLeïla TricotMohamad ZaidanAlain WynckelJulien ZuberMoglie Le QuintrecVéronique Frémeaux- BacchiSophie Chauvet
Published in: Clinical journal of the American Society of Nephrology : CJASN (2023)
In our cohort, 17% of C3 glomerulopathy/Ig-MPGN cases were associated with rare variants in the CFH, CFI, or C3 genes. In the majority of cases, a quantitative deficiency in factor H or factor I is identified. The presence of a rare variant was associated with poor kidney survival.
Keyphrases
  • copy number
  • genome wide
  • genome wide identification
  • high resolution
  • dna methylation
  • gene expression
  • transcription factor
  • replacement therapy
  • genome wide analysis
  • smoking cessation