Alteration in Cerebral Metabolism in a Rodent Model of Acute Sub-lethal Cyanide Poisoning.

Oladunni AlomajaFrances S ShoferJohn C GreenwoodSarah PielCarly ClaymanClementina MesarosShih-Han KaoSamuel S ShinJohannes K EhingerTodd J KilbaughDavid H Jang

Published in: Journal of medical toxicology : official journal of the American College of Medical Toxicology (2023)

The key finding demonstrates mitochondrial dysfunction in brain tissue that corresponds with a decrease in mitochondrial function, ATP concentrations, and an elevated LPR indicating brain dysfunction at a sub-lethal dose of cyanide.

Keyphrases

white matter
resting state
cerebral ischemia
liver failure
fluorescent probe
oxidative stress
respiratory failure
aortic dissection