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Changes in Synaptic Transmission and Long-term Potentiation Induction as a Possible Mechanism for Learning Disability in an Animal Model of Multiple Sclerosis.

Ghasem MosayebiMohammad Reza SoleymanMostafa KhaliliMasoumeh MoslehMohammad Reza Palizvan
Published in: International neurourology journal (2016)
In conclusion, influence of inflammation-triggered mechanisms on synaptic transmission may explain the negative effect of EAE on learning abilities in rats.
Keyphrases
  • multiple sclerosis
  • prefrontal cortex
  • oxidative stress
  • white matter