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Thiol-Disulfide Homeostasis, Serum Ferroxidase Activity, and Serum Ischemia Modified Albumin Levels in Neonatal Jaundice.

Ismail TopalCuma MertogluIlknur Surucu KaraGulsah SiranliGamze GokÖzcan Erel
Published in: Fetal and pediatric pathology (2019)
Altered thiol/disulfide homeostasis in the favor of disulfide indicates augmented oxidative stress in jaundiced term infants. The lack of alteration in ferroxidase or IMA levels suggests these latter alterations take more time or more severe oxidative stress to become altered or are not as sensitive as the thiol/disulfide ratio to detect oxidative stress states.
Keyphrases
  • oxidative stress
  • dna damage
  • diabetic rats
  • ischemia reperfusion injury
  • induced apoptosis
  • preterm infants
  • early onset
  • signaling pathway
  • preterm birth