Synaptic density marker SV2A is reduced in schizophrenia patients and unaffected by antipsychotics in rats.
Ellis Chika OnwordiElise F HalffThomas WhitehurstAyla MansurMarie-Caroline CotelLisa WellsHannah CreeneyDavid BonsallMaria RogdakiEkaterina ShatalinaTiago Reis MarquesEugenii A RabinerRoger N GunnSridhar NatesanAnthony Christopher VernonOliver D HowesPublished in: Nature communications (2020)
Synaptic dysfunction is hypothesised to play a key role in schizophrenia pathogenesis, but this has not been tested directly in vivo. Here, we investigated synaptic vesicle glycoprotein 2A (SV2A) levels and their relationship to symptoms and structural brain measures using [11C]UCB-J positron emission tomography in 18 patients with schizophrenia and 18 controls. We found significant group and group-by-region interaction effects on volume of distribution (VT). [11C]UCB-J VT was significantly lower in the frontal and anterior cingulate cortices in schizophrenia with large effect sizes (Cohen's d = 0.8-0.9), but there was no significant difference in the hippocampus. We also investigated the effects of antipsychotic drug administration on SV2A levels in Sprague-Dawley rats using western blotting, [3H]UCB-J autoradiography and immunostaining with confocal microscopy, finding no significant effects on any measure. These findings indicate that there are lower synaptic terminal protein levels in schizophrenia in vivo and that antipsychotic drug exposure is unlikely to account for them.
Keyphrases
- bipolar disorder
- positron emission tomography
- prefrontal cortex
- computed tomography
- end stage renal disease
- functional connectivity
- drug administration
- chronic kidney disease
- resting state
- ejection fraction
- prognostic factors
- oxidative stress
- south africa
- cognitive impairment
- patient reported outcomes
- high resolution
- adverse drug