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Eicosapentaenoic acid protects cardiomyoblasts from lipotoxicity in an autophagy-dependent manner.

Hsiu-Ching HsuSin-Jin LiChing-Yi ChenMing-Fong Chen
Published in: Cell biology and toxicology (2017)
EPA reduces lipotoxicity-induced cardiomyoblast apoptosis through its effects on autophagy.
Keyphrases
  • endoplasmic reticulum stress
  • cell death
  • oxidative stress
  • diabetic rats
  • cell cycle arrest
  • signaling pathway
  • high glucose
  • drug induced
  • endothelial cells
  • pi k akt