LY6K depletion modulates TGF-β and EGF signaling.

Sujeong ParkDoyeon ParkSora HanGa Eun ChungSujung SohHye In KaHyun Jeong JooYoung Yang

Published in: Cancer medicine (2023)

Our study demonstrates the key role of LY6K in both clathrin- and CAV-1-mediated endocytic pathways regulated by TGF-β and EGF, and it suggests a correlation between LY6K overexpression in cervical cancer cells and poor overall survival.

Keyphrases

transforming growth factor
growth factor
cell proliferation
epithelial mesenchymal transition
free survival